FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
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miR‑192 is upregulated in T1DM, regulates pancreatic β‑cell development and inhibits insulin secretion through suppressing GLP‑1 expression
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FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
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Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016)
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
Figure S1, FGFR2 copy gain and overexpression in H716 cells A - ppt download
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Uncoupling protein 2 regulates glucagon-like peptide-1 secretion in L-cells
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The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
Uncoupling protein 2 regulates glucagon-like peptide-1 secretion in L-cells
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Extracellular matrix components induce endocrine differentiation in vitro in NCI-H716 cells. - Abstract - Europe PMC
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
NCI-H716 cells as a model for endocrine differentiation in colorectal cancer
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FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
Microorganisms | Free Full-Text | Roseburia intestinalis Modulates PYY Expression in a New a Multicellular Model including Enteroendocrine Cells
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
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